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- W2006283019 abstract "Gastroesophageal reflux (GER) is a potential trigger of asthma. Approximately 77% of asthmatics report heartburn. GER is a risk factor for asthma-related hospitalization and oral steroid burst use. Asthmatics may be predisposed to GER development because of a high prevalence of hiatal hernia and autonomic dysregulation and an increased pressure gradient between the abdominal cavity and the thorax, over-riding the lower esophageal sphincter pressure barrier. Asthma medications may potentiate GER. Potential mechanisms of esophageal acid-induced bronchoconstriction include a vagally mediated reflex, local axonal reflexes, heightened bronchial reactivity, and microaspiration, all resulting in neurogenic inflammation. Anti-reflux therapy improves asthma symptoms in approximately 70% of asthmatics with GER. A 3-month empiric trial of twice-daily proton pump inhibitor given 30 to 60 minutes before breakfast and dinner can identify asthmatics who have GER as a trigger of their asthma." @default.
- W2006283019 created "2016-06-24" @default.
- W2006283019 creator A5002269164 @default.
- W2006283019 date "2005-02-01" @default.
- W2006283019 modified "2023-10-16" @default.
- W2006283019 title "Gastroesophageal reflux: a potential asthma trigger" @default.
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- W2006283019 doi "https://doi.org/10.1016/j.iac.2004.09.006" @default.
- W2006283019 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/15579368" @default.
- W2006283019 hasPublicationYear "2005" @default.
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