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- W2006756811 abstract "Most neuroscientists now acknowledge that there is more to the brain than neurons. A flurry of studies in the past decade have documented the importance of glia in complex processes that were previously believed to be strictly neuronal.1 The majority of these studies have pointed to a key role of astrocytic Ca2+ signaling in the modulation of synaptic transmission. In fact, Ca2+ signaling in electrically silent astrocytes can in many ways be compared with electrical excitability in neurons: Astrocytes integrate multiple inputs into dynamic Ca2+ signals and in turn modulate the activity of surrounding neurons. Perhaps, the most interesting discovery is that astrocytic Ca2+ signaling appears to be intimately involved in neural homeostatic regulation. For example, Ca2+ signaling in brain stem astrocytes may help to control breathing: A reduction in extracellular pH leads to astrocytic Ca2+-dependent ATP release. ATP binds to purinergic (ATP) receptors on neighboring respiratory neurons, depolarizing them and resulting in an increase in respiration.2" @default.
- W2006756811 created "2016-06-24" @default.
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- W2006756811 date "2012-12-05" @default.
- W2006756811 modified "2023-10-17" @default.
- W2006756811 title "A Link between Glial Ca2+ Signaling and Hypoxia in Aging?" @default.
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- W2006756811 doi "https://doi.org/10.1038/jcbfm.2012.182" @default.
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