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- W2006992169 endingPage "187" @default.
- W2006992169 startingPage "179" @default.
- W2006992169 abstract "Lipid and glycolipid mediators are important messengers of the adaptive responses to stress, including apoptosis. In mammalian cells, the intracellular accumulation of ganglioside GD3, an acidic glycosphingolipid, contributes to mitochondrial damage, a crucial event during the apoptopic program. GD3 is a minor ganglioside in most normal tissues. Its expression increases during development and in pathological conditions such as cancer and neurodegenerative disorders. Intriguingly, GD3 can mediate additional biological events such as cell proliferation and differentiation. These diverse and opposing effects indicate that tightly regulated mechanisms, including 9-O-acetylation, control GD3 function, by affecting intracellular levels, localization and structure of GD3, and eventually dictate biological outcomes and cell fate decisions." @default.
- W2006992169 created "2016-06-24" @default.
- W2006992169 creator A5005762171 @default.
- W2006992169 creator A5076073255 @default.
- W2006992169 date "2002-12-01" @default.
- W2006992169 modified "2023-09-23" @default.
- W2006992169 title "GD3 ganglioside and apoptosis" @default.
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