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• W2007023970 abstract "To investigate the role of neutrophils in experimental cerebral malaria (ECM), in a previous study we found that early neutrophil depletion prevented the development of ECM and down regulated the expression of Th1 cytokines in the brain. To further clarify the mechanisms responsible for these findings, in the present study, using RT-PCR, we examined the expression of cytokine and chemokine mRNAs in neutrophils and macrophages after PbA infection. We found that, after infection, neutrophils not only expressed cytokines IL-2, IL-12p40, IL-18, IFN-γ and TNF-α mRNAs, but also mRNAs for Th1 chemoattractive chemokines, monokine-induced by IFN-γ (MIG), macrophage-inflammatory protein-1α (MIP-1α) and IFN-γ inducible protein-10 (IP-10). Neutrophil depletion down regulated the expression of IL-18 and MIG mRNAs in macrophages, but did not affect the expression of IFN-γ, TNF-α, MIP-1α and IP-10 mRNAs. Therefore, this study confirms our hypothesis that neutrophils may play a role in the pathogenesis of ECM via their expression of cytokines or chemokines." @default.
• W2007023970 created "2016-06-24" @default.
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• W2007023970 date "2001-07-01" @default.
• W2007023970 modified "2023-10-03" @default.
• W2007023970 title "Cytokine and chemokine mRNA expression in neutrophils from CBA/NSlc mice infected with Plasmodium berghei ANKA that induces experimental cerebral malaria" @default.
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• W2007023970 doi "https://doi.org/10.1016/s1383-5769(01)00063-0" @default.
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