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• W2007083175 abstract "Substantial evidence supports a role for the renin–angiotensin system (RAS) in the regulation of metabolic function, but an apparent paradox exists where genetic or pharmacological inhibition of the RAS occasionally has similar physiological effects as chronic angiotensin infusion. Similarly, while RAS targeting in animal models has robust metabolic consequences, effects in humans are more subtle. Here, we review the data supporting a role for the RAS in metabolic rate regulation and propose a model where the local brain RAS works in opposition to the peripheral RAS, thus helping to explain the paradoxically similar effects of RAS supplementation and inhibition. Selectively modulating the peripheral RAS or brain RAS may thus provide a more effective treatment paradigm for obesity and obesity-related disorders." @default.
• W2007083175 created "2016-06-24" @default.
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• W2007083175 creator A5085916275 @default.
• W2007083175 date "2012-04-12" @default.
• W2007083175 modified "2023-09-27" @default.
• W2007083175 title "Metabolic rate regulation by the renin–angiotensin system: brain vs. body" @default.
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• W2007083175 doi "https://doi.org/10.1007/s00424-012-1096-9" @default.
• W2007083175 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3569511" @default.
• W2007083175 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/22491893" @default.
• W2007083175 hasPublicationYear "2012" @default.
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