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• W2007138966 abstract "Significance Cell survival after DNA damage relies on DNA repair, the abrogation of which causes genomic instability and development of cancer. DNA double-strand breaks are lesions induced by ionizing radiation (IR) and can be efficiently repaired by DNA homologous recombination, a system that requires RAD51 recombinase (RAD51). Here we show that overexpression of miR-155 in human breast cancer cells reduces the levels of RAD51 and affects the cellular response to IR. High miR-155 levels were associated with lower RAD51 expression and with better overall survival of patients in a large series of triple-negative breast cancers. Testing triple-negative breast cancer patients for miR-155 expression may be a useful prognostic tool to identify who will benefit from an IR-based therapeutic approach." @default.
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• W2007138966 date "2014-03-10" @default.
• W2007138966 modified "2023-10-15" @default.
• W2007138966 title "Protective role of miR-155 in breast cancer through <i>RAD51</i> targeting impairs homologous recombination after irradiation" @default.
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• W2007138966 doi "https://doi.org/10.1073/pnas.1402604111" @default.
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