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• W2007193090 abstract "<h2>Abstract</h2> Transcriptional dysregulation is a major pathological feature of Huntington's disease (HD). The goal of this study was to understand how p65/RelA co-regulated genes, specifically those of the cytokine and endocannabinoid systems, were affected in HD. p65/RelA levels were lower in human HD tissue and R6/2 HD mice, as were the levels of the type 1 cannabinoid receptor (CB₁), IL-1β, IL-8, CCL5, GM-CSF, MIP-1β, and TNFα, all of which may be regulated by p65/RelA. Activation of p65/RelA restored CB₁ and CCL5 expression in ST<i>Hdh</i> cell models of HD. Therefore, p65/RelA activation may normalize the expression of some genes in HD." @default.
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• W2007193090 date "2014-02-01" @default.
• W2007193090 modified "2023-09-25" @default.
• W2007193090 title "The cytokine and endocannabinoid systems are co-regulated by NF-κB p65/RelA in cell culture and transgenic mouse models of Huntington's disease and in striatal tissue from Huntington's disease patients" @default.
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• W2007193090 doi "https://doi.org/10.1016/j.jneuroim.2013.12.008" @default.
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