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- W2007272589 endingPage "e974448" @default.
- W2007272589 startingPage "e974448" @default.
- W2007272589 abstract "Endothelial cells (EC) form a semi-permeable barrier between the interior space of blood vessels and the underlying tissues. In acute lung injury (ALI) the EC barrier is weakened leading to increased vascular permeability. It is widely accepted that EC barrier integrity is critically dependent upon intact cytoskeletal structure and cell junctions. Edemagenic agonists, like thrombin or endotoxin lipopolysaccharide (LPS), induced cytoskeletal rearrangement, and EC contractile responses leading to disruption of intercellular contacts and EC permeability increase. The highly clinically-relevant cytoskeletal mechanisms of EC barrier dysfunction are currently under intense investigation and will be described and discussed in the current review." @default.
- W2007272589 created "2016-06-24" @default.
- W2007272589 creator A5071655149 @default.
- W2007272589 creator A5075667272 @default.
- W2007272589 creator A5084110035 @default.
- W2007272589 date "2014-12-31" @default.
- W2007272589 modified "2023-10-10" @default.
- W2007272589 title "Cytoskeletal mechanisms regulating vascular endothelial barrier function in response to acute lung injury" @default.
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