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- W2007489093 endingPage "1015" @default.
- W2007489093 startingPage "1003" @default.
- W2007489093 abstract "Helicobacter pylori infection is the major etiological factor of gastric carcinoma. This disease is the result of a long, multistep, and multifactorial process, which occurs only in a small proportion of patients infected with H. pylori. Gastric carcinoma development is influenced by host genetic susceptibility factors, environmental factors, and H. pylori virulence. H. pylori is genetically highly variable, and variability that affects H. pylori virulence factors may be useful to identify strains with different degrees of pathogenicity. This review will focus on VacA and CagA that have polymorphic regions that impact their functional properties. The characterization of H. pylori vacA and cagA-associated could be useful for identifying patients at highest risk of disease, who could be offered H. pylori eradication therapy and who could be included in programs of more intensive surveillance in an attempt to reduce gastric carcinoma incidence." @default.
- W2007489093 created "2016-06-24" @default.
- W2007489093 creator A5018172201 @default.
- W2007489093 creator A5043808791 @default.
- W2007489093 creator A5052256882 @default.
- W2007489093 date "2014-12-01" @default.
- W2007489093 modified "2023-10-09" @default.
- W2007489093 title "Clinical relevance of Helicobacter pylori vacA and cagA genotypes in gastric carcinoma" @default.
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