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Matches in SemOpenAlex for { <https://semopenalex.org/work/W2007998684> ?p ?o ?g. }

• W2007998684 endingPage "993" @default.
• W2007998684 startingPage "990" @default.
• W2007998684 abstract "In normal human cells, damage due to ultraviolet light is preferentially removed from active genes by nucleotide excision repair (NER) in a transcription-coupled repair (TCR) process that requires the gene products defective in Cockayne syndrome (CS). Oxidative damage, including thymine glycols, is shown to be removed by TCR in cells from normal individuals and from xeroderma pigmentosum (XP)-A, XP-F, and XP-G patients who have NER defects but not from XP-G patients who have severe CS. Thus, TCR of oxidative damage requires an XPG function distinct from its NER endonuclease activity. These results raise the possibility that defective TCR of oxidative damage contributes to the developmental defects associated with CS." @default.
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• W2007998684 date "1997-02-14" @default.
• W2007998684 modified "2023-09-26" @default.
• W2007998684 title "Defective Transcription-Coupled Repair of Oxidative Base Damage in Cockayne Syndrome Patients from XP Group G" @default.
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• W2007998684 doi "https://doi.org/10.1126/science.275.5302.990" @default.
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