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- W2008105473 abstract "Stimulation of phosphoinositide hydrolysis in myocardium from autoimmune myocarditis mice by ThEA and histamine was assayed. Myocardium from autoimmune heart, but not the normal forms, specifically increased phosphoinositide turnover in the presence of histaminergic agonists. This increment was blocked by a specific H1 antagonist mepyramine and to the same extent by the phospholipase C inhibitor NCDC. By using a binding assay H1 histaminergic receptors were detected in autoimmune heart membrane preparations, but this was not observed in normal heart. These data suggest that autoimmune myocardium expressed a functional H1 receptor that could involve a distinctive mechanism operating in the disease." @default.
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- W2008105473 date "1993-01-01" @default.
- W2008105473 modified "2023-10-16" @default.
- W2008105473 title "Phosphoinositide hydrolysis mediated by H1 receptors in autoimmune myocarditis mice" @default.
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- W2008105473 doi "https://doi.org/10.1155/s0962935193000444" @default.
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