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- W2008181873 abstract "Helicobacter pylori infection represents the strongest known risk factor for the development of gastric cancer. The vacuolating cytotoxin (VacA) plays a key role in disease pathogenesis by exerting pleiotrophic effects on the host. One effect of acute VacA exposure is the induction of autophagy. However, prolonged exposure to the toxin disrupts autophagy by preventing maturation of the autolysosome. Novel insights into the mechanism and consequences of this phenomenon have emerged, but many aspects remain largely unknown. Current evidence supports a scenario in which H. pylori-suppressed autophagy facilitates intracellular survival and persistence of the pathogen, while also generating an environment favoring carcinogenesis." @default.
- W2008181873 created "2016-06-24" @default.
- W2008181873 creator A5071012946 @default.
- W2008181873 creator A5083599998 @default.
- W2008181873 date "2013-11-01" @default.
- W2008181873 modified "2023-09-25" @default.
- W2008181873 title "Modulation of autophagy by Helicobacter pylori and its role in gastric carcinogenesis" @default.
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- W2008181873 doi "https://doi.org/10.1016/j.tim.2013.09.004" @default.
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