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- W2008336230 endingPage "233" @default.
- W2008336230 startingPage "227" @default.
- W2008336230 abstract "The lymphotoxin (LT)-pathway is a unique constituent branch of the Tumor Necrosis Superfamily (TNFSF). Use of LT is a critical mechanism by which fetal innate lymphoid cells regulate lymphoid organogenesis. Within recent years, adult innate lymphoid cells have been discovered to utilize this same pathway to regulate IL-22 and IL-23 production for host defense. Notably, genetic studies have linked polymorphisms in the genes encoding LTα to several phenotypes contributing to metabolic syndrome. The role of the LT-pathway may lay the foundation for a bridge between host immune response, microbiota, and metabolic syndrome. The contribution of the LT-pathway to innate lymphoid cell function and metabolic syndrome will be visited in this review." @default.
- W2008336230 created "2016-06-24" @default.
- W2008336230 creator A5022331042 @default.
- W2008336230 creator A5035676180 @default.
- W2008336230 date "2014-04-01" @default.
- W2008336230 modified "2023-10-18" @default.
- W2008336230 title "Lymphotoxin organizes contributions to host defense and metabolic illness from innate lymphoid cells" @default.
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- W2008336230 doi "https://doi.org/10.1016/j.cytogfr.2013.12.007" @default.
- W2008336230 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3999173" @default.