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• W2008491373 abstract "In eukaryotes, homologous recombination is an important pathway for the repair of DNA double-strand breaks. We have studied this process in living cells in the yeast Saccharomyces cerevisiae using Rad52 as a cell biological marker. In response to DNA damage, Rad52 redistributes itself and forms foci specifically during S phase. We have shown previously that Rad52 foci are centers of DNA repair where multiple DNA double-strand breaks colocalize. Here we report a correlation between the timing of Rad52 focus formation and modification of the Rad52 protein. In addition, we show that the two ends of a double-strand break are held tightly together in the majority of cells. Interestingly, in a small but significant fraction of the S phase cells, the two ends of a break separate suggesting that mechanisms exist to reassociate and align these ends for proper DNA repair." @default.
• W2008491373 created "2016-06-24" @default.
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• W2008491373 date "2003-09-12" @default.
• W2008491373 modified "2023-09-24" @default.
• W2008491373 title "Cell Cycle-Regulated Centers of DNA Double-Strand Break Repair" @default.
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• W2008491373 doi "https://doi.org/10.4161/cc.2.5.483" @default.
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