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• W2008654911 abstract "Besides locomotion, organ protection, and calcium-phosphorus homeostasis, the three classical functions of the skeleton, bone remodeling affects energy metabolism through uncarboxylated osteocalcin, a recently discovered hormone secreted by osteoblasts. This review traces how energy metabolism affects osteoblasts through the central control of bone mass involving leptin, serotoninergic neurons, the hypothalamus, and the sympathetic nervous system. Next, the role of osteocalcin (insulin secretion, insulin sensitivity, and pancreas β-cell proliferation) in the regulation of energy metabolism is described. Then, the connections between insulin signaling on osteoblasts and the release of uncarboxylated osteocalcin during osteoclast bone resorption through osteoprotegerin are reported. Finally, the understanding of this new bone endocrinology will provide some insights into bone, kidney, and energy metabolism in patients with chronic kidney disease." @default.
• W2008654911 created "2016-06-24" @default.
• W2008654911 creator A5084591362 @default.
• W2008654911 date "2011-04-01" @default.
• W2008654911 modified "2023-09-30" @default.
• W2008654911 title "Bone: from a reservoir of minerals to a regulator of energy metabolism" @default.
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• W2008654911 doi "https://doi.org/10.1038/ki.2011.25" @default.
• W2008654911 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3257050" @default.
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