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- W2008982878 abstract "THE normal cardiac pump adapts remarkably well to peripheral demands, which vary continuously and are often extreme. By shortening its sarcomeres further and more forcefully, it quickly delivers increased flow at high pressures, but in so doing it subjects its wall to considerable systolic stress. How do the thinner surfaces of this hollow muscular pump normally avoid systolic bulging, and why may they rupture when infarcted?Normal cardiac muscle consists largely of myofibrils and mitochondria.1 At rest its compliance is midway between that of collagen and that of elastin. With activation and formation of rigid actin–myosin cross-links, it becomes 10 . . ." @default.
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- W2008982878 date "1983-09-01" @default.
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- W2008982878 title "Infarction and Rupture of the Heart" @default.
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- W2008982878 doi "https://doi.org/10.1056/nejm198309013090910" @default.
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