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- W2009062406 endingPage "1040" @default.
- W2009062406 startingPage "1032" @default.
- W2009062406 abstract "Among the cytokines linked to inflammation-associated cancer, interleukin (IL)-6 drives many of the cancer 'hallmarks' through downstream activation of the Janus kinase/signal transducer and activator of transcription 3 (JAK/STAT3) signaling pathway. Additionally, dysregulation of the interleukin (IL)-6-mediated JAK/STAT3 signaling pathway is closely related to the development of diverse human solid tumors including colorectal cancer (CRC). On this basis, modulation of the IL-6/JAK/STAT3 signaling pathway is currently being widely explored to develop novel therapies for CRC. The present review details the mechanisms and roles of the IL-6/JAK/STAT3 pathway in CRC, describes current therapeutic strategies, and the search for potential therapeutic approaches to treat CRC." @default.
- W2009062406 created "2016-06-24" @default.
- W2009062406 creator A5069226662 @default.
- W2009062406 creator A5088116210 @default.
- W2009062406 date "2014-01-15" @default.
- W2009062406 modified "2023-10-14" @default.
- W2009062406 title "The IL-6/JAK/STAT3 pathway: Potential therapeutic strategies in treating colorectal cancer" @default.
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- W2009062406 doi "https://doi.org/10.3892/ijo.2014.2259" @default.
- W2009062406 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24430672" @default.
- W2009062406 hasPublicationYear "2014" @default.
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