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- W2009067699 endingPage "550" @default.
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- W2009067699 abstract "We propose a model where autoimmunity can be viewed as a dynamic system driven by opposite vectors IFN-α/β and TNF. These cytokines drive differentiation of distinct types of DCs, TNF-DCs, or IFN-DCs, which present different antigens leading to distinct autoimmune responses. When balanced, both cytokines synergize in protective immunity. When one of the cytokines prevails, autoimmunity occurs, Type I interferons (IFN-α/β) playing a major role in systemic lupus erythematosus (SLE) and TNF playing a major role in rheumatoid arthritis. This model complements the Type 1/Type 2 paradigm. Therefore, immunity can be viewed as a dynamic system driven by two sets of opposite vectors: IFN-α/β/TNF and IFN-γ/IL-4." @default.
- W2009067699 created "2016-06-24" @default.
- W2009067699 creator A5002962815 @default.
- W2009067699 creator A5041412546 @default.
- W2009067699 creator A5058800155 @default.
- W2009067699 date "2004-05-01" @default.
- W2009067699 modified "2023-10-03" @default.
- W2009067699 title "Autoimmunity through Cytokine-Induced Dendritic Cell Activation" @default.
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