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• W2009097329 endingPage "94" @default.
• W2009097329 startingPage "87" @default.
• W2009097329 abstract "Innate sensors of viral infection detect viral products and initiate the signal cascades that lead to the antiviral response. Several proteins have been identified to play a role in this process, mostly members of the Toll-like receptor and retinoic acid-inducible gene I-like receptor families. These receptors have been demonstrated to function in part by recognizing a diverse yet unique repertoire of nucleic acid substrates. Upon recognition of their ligands, these sensors activate distinct signaling pathways that lead to the secretion of type I interferon and inflammatory cytokines. It remains to be seen, however, if these sensors are redundant or whether each serves a unique function. In this work, we review the current knowledge of viral sensors, speculate on how they may function in vivo, and explore the potential reasons for their diversity." @default.
• W2009097329 created "2016-06-24" @default.
• W2009097329 creator A5055691289 @default.
• W2009097329 creator A5081030853 @default.
• W2009097329 date "2008-12-19" @default.
• W2009097329 modified "2023-10-16" @default.
• W2009097329 title "Viral sensors: diversity in pathogen recognition" @default.
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• W2009097329 doi "https://doi.org/10.1111/j.1600-065x.2008.00726.x" @default.
• W2009097329 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/7165970" @default.
• W2009097329 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19120478" @default.

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