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• W2009538412 abstract "ABSTRACT A double-guanine-insertion mutation within a run of guanines in the herpes simplex virus gene encoding thymidine kinase (TK) was previously found in an acyclovir-resistant clinical isolate. This mutation was engineered into strain KOS, and stocks were generated from single plaques. Plaque autoradiography revealed that most plaques in such stocks exhibited low levels of TK activity, while ∼3% of plaques exhibited high levels of TK activity, indicating a remarkably high frequency of phenotypic reversion. This virus was able to reactivate from latency in mouse ganglia; a fraction of the reactivating virus expressed a high level of TK activity due to an additional G insertion, suggesting that the observed genetic instability contributed to pathogenicity." @default.
• W2009538412 created "2016-06-24" @default.
• W2009538412 creator A5061147470 @default.
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• W2009538412 date "2003-02-01" @default.
• W2009538412 modified "2023-09-30" @default.
• W2009538412 title "High-Frequency Phenotypic Reversion and Pathogenicity of an Acyclovir-Resistant Herpes Simplex Virus Mutant" @default.
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• W2009538412 doi "https://doi.org/10.1128/jvi.77.3.2282-2286.2003" @default.
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