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- W2009758435 startingPage "565" @default.
- W2009758435 abstract "Secondary degeneration occurs commonly in the central nervous system after traumatic injuries and following acute and chronic diseases, including glaucoma. A constellation of mechanisms have been shown to be associated with secondary degeneration including apoptosis, necrosis, autophagy, oxidative stress, excitotoxicity, derangements in ionic homeostasis and calcium influx. Glial cells, such as microglia, astrocytes and oligodendrocytes, have also been demonstrated to take part in the process of secondary injury. Partial optic nerve transection is a useful model which was established about 13 years ago. The merit of this model compared with other optic nerve injury models used for glaucoma study, including complete optic nerve transection model and optic nerve crush model, is the possibility to separate primary degeneration from secondary degeneration in location. Therefore, it provides a good tool for the study of secondary degeneration. This review will focus on the research progress of the mechanisms of secondary degeneration using partial optic nerve transection model." @default.
- W2009758435 created "2016-06-24" @default.
- W2009758435 creator A5005434770 @default.
- W2009758435 creator A5019685354 @default.
- W2009758435 creator A5020504300 @default.
- W2009758435 creator A5052529067 @default.
- W2009758435 creator A5074047749 @default.
- W2009758435 date "2014-01-01" @default.
- W2009758435 modified "2023-10-16" @default.
- W2009758435 title "Mechanisms of secondary degeneration after partial optic nerve transection" @default.
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