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• W2010082296 abstract "Yohimbine (2.5 or 4 mg/kg) reduced the percentage of open arm entries and the percentage of time spent on the open arms displayed by rats on an elevated plus-maze indicating anxiogenic activity. These effects were reversed by the α2-adrenoceptor agonist clonidine (0.01 mg/kg) and by the dopamine receptor agonist apomorphine (0.57 mg/kg). The following failed to reverse the effects of yohimbine: the selective α2-adrenoceptor agonists, guanfacine (0.25 and 1 mg/kg), B-HT920 (0.025 and 0.1 mg/kg), B-HT933 (1 and 10 mg/kg); the β-blocker propanolol (2.5 and 10 mg/kg); the α1-adrenoceptor agonist phenylephrine; the D1 agonist SK&F 38393 (5 and 10 mg/kg) and the D2 agonist LY 171555 (0.5 and 1 mg/kg). Therefore, it is unlikely that activity at only the α1, α2, β, D1 or D2 sites can entirely account for the anxiogenic actions of yohimbine in the elevated plus-maze. Evidence that clonidine affects the dopaminergic system and that apomorphine affects the noradrenergic system suggests that yohimbine may produce its anxiogenic response by activity on both the noradrenergic and dopaminergic systems." @default.
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• W2010082296 date "1989-01-01" @default.
• W2010082296 modified "2023-10-11" @default.
• W2010082296 title "Yohimbine's anxiogenic action: Evidence for noradrenergic and dopaminergic sites" @default.
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• W2010082296 doi "https://doi.org/10.1016/0091-3057(89)90225-6" @default.
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