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• W2010227856 abstract "It is well established that coeliac disease with mucosal inflammation, small bowel villous atrophy and crypt hyperplasia may develop by time [ [1] Ferguson A. Arranz E. O’Mahony S. Clinical and pathological spectrum of coeliac disease – active, silent, latent, potential. Gut. 1993; 34: 150-151 Crossref PubMed Scopus (392) Google Scholar ]. This has been clearly shown in individuals positive for serum endomysial or transglutaminase antibodies and normal small bowel mucosa [ 2 Collin P. Helin H. Mäki M. Hällström O. Karvonen A.-L. Follow-up of patients positive in reticulin and gliadin antibody tests with normal small bowel biopsy findings. Scand J Gastroenterol. 1993; 28: 595-598 Crossref PubMed Scopus (148) Google Scholar , 3 Salmi T.T. Collin P. Jarvinen O. Haimila K. Partanen J. Laurila K. et al. Immunoglobulin A autoantibodies against transglutaminase 2 in the small intestinal mucosa predict forthcoming coeliac disease. Aliment Pharmacol Ther. 2006; 24: 541-552 Crossref PubMed Scopus (133) Google Scholar ], whereas the data are scanty in those with negative coeliac antibodies. However, evidence suggests initially seronegative people may become seropositive and subsequently develop coeliac disease. This has been shown in especially type 1 diabetes mellitus, a well-known risk group for coeliac disease: Mäki et al. [ [4] Mäki M. Huupponen T. Holm K. Hällström O. Seroconversion of reticulin autoantibodies predicts coeliac disease in insulin dependent diabetes mellitus. Gut. 1995; 36: 239-242 Crossref PubMed Scopus (122) Google Scholar ] observed such a seroconversion for endomysial (reticulin) antibodies in 11 (4.6%) out of 238 and Saukkonen et al. [ [5] Saukkonen T. Savilahti E. Reijonen H. Ilonen J. Tuomilehto-Wolf E. Åkerblom H.K. Coeliac disease: Frequent occurrence after clinical onset of insulin-dependent diabetes mellitus. Diabet Med. 1996; 13: 464-470 Crossref PubMed Scopus (131) Google Scholar ] in 9 (1.2%) out of 776 diabetic children, with a subsequent demonstration of small bowel villous atrophy. A similar seroconversion and mucosal atrophy has been shown by Barera et al. [ [6] Barera G. Bonfanti R. Viscardi M. Bazigaluppi E. Calori G. Meschi F. et al. Occurrence of celiac disease after onset of type 1 diabetes: A 6-year prospective longitudinal study. Pediatrics. 2002; 109: 952-954 Crossref PubMed Scopus (226) Google Scholar ] and Crone et al. [ [7] Crone J. Rami B. Huber W.D. Granditsch G. Schober E. Prevalence of celiac disease and follow-up of EMA in children and adolescents with type I diabetes mellitus. J Pediatr Gastroenterol Nutr. 2003; 37: 67-71 Crossref PubMed Scopus (65) Google Scholar ]. Thus, the development of coeliac disease by time is not extremely uncommon in type 1 diabetes. How about other risk groups for coeliac disease, and how about patients with borderline small bowel histological findings?" @default.
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• W2010227856 title "Serologic screening for coeliac disease in risk groups: Is once in the lifetime enough?" @default.
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