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- W2010435751 abstract "See related article, pages 250–255. Time plays a major role in acute stroke treatment. Millions of neurons are lost each minute in acute ischemic stroke,1 and the efficacy of intravenous thrombolysis is related to time from symptom onset with earlier treatment being more effective.2,3 Moreover, knowledge of the time of symptom onset is a prerequisite for treatment with intravenous thrombolysis. This information, however, is lacking in a large proportion of patients with acute stroke. In 14% to 27% of patients, stroke symptoms occur during sleep,4–6 and this large group of patients is a priori excluded from the only specific and effective treatment available for acute stroke. This dissatisfying situation has raised interest in imaging surrogate markers of lesion age, which might help identify patients presenting within a time window making them eligible for thrombolytic treatment.6In this issue of Stroke, Ebinger et al report on a study on the relation between the visibility of acute ischemic lesions in fluid-attenuated inversion recovery (FLAIR) MRI and lesion age.7 Stroke MRI lends itself to be used as a surrogate marker of lesion age with signal changes in different MRI sequences taking a different course during the first hours of ischemia. Although diffusion-weighted imaging (DWI) allows for the detection of signal changes resulting from cytotoxic edema within minutes, the T2 relaxation time increases continuously during the first hours of stroke.8 This …" @default.
- W2010435751 created "2016-06-24" @default.
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- W2010435751 date "2010-02-01" @default.
- W2010435751 modified "2023-09-27" @default.
- W2010435751 title "We Are on the Clock" @default.
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- W2010435751 doi "https://doi.org/10.1161/strokeaha.109.570085" @default.
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