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• W2010488262 abstract "Experimental and clinical evidence supports the notion that the Epstein-Barr virus (EBV) can become oncogenic in immune-deficient hosts. Renal transplant recipients and children with inherited immune deficiency are at high risk for lymphomagenesis. Defective immune responses to EBV permit persistence of polyclonal proliferation of B cells. Conversion from polyclonal to monoclonal proliferation is probably the result of a cytogenetic error in a B cell. This hypothesis can be tested prospectively in patients with immune deficiency by immunological, EBV, and genetic studies." @default.
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• W2010488262 date "1980-02-01" @default.
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• W2010488262 title "EPSTEIN-BARR-VIRUS-INDUCED ONCOGENESIS IN IMMUNE-DEFICIENT INDIVIDUALS" @default.
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• W2010488262 doi "https://doi.org/10.1016/s0140-6736(80)90792-8" @default.
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