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- W2010502097 abstract "Abstract Male weanling Lewis rats were fed diets either adequate (5.6 μg/g) or low (0.6 μg/g) in copper, either and libitum or in restricted amounts for 14–70 days. Measurements were made of food intake; growth rate; hematocrits; Cu and Fe concentrations of plasma or serum; ceruloplasmin activity of plasma or serum; plasma cholesterol and whole blood lactate concentrations; cytochrome c oxidase activity of spleen lymphoid cells (SLC); and proliferation of SLC, cervical lymph node cells (CLNC) and plastic nonadherent-SLC (NASLC) following stimulation with concanavalin-A (Con-A). Cu-deficient (CD) rats exhibited upon completion of dietary regimen days: 14–18, reduced hematocrits, plasma Cu and Fe, plasma ceruloplasmin activity and cytochrome c oxidase activity of SLC; 28–32, elevated plasma cholesterol and blood lactate, decreased proliferation of Con-A stimulated SLC, but increased proliferation of Con-A stimulated CLNC; and 42–46, reduced food intake and growth. Suppressed proliferation of Con-A stimulated SLC from CD rats was not alleviated following removal of plastic-adherent lymphoid cells from the SLC suspension. Equivalent proliferation was exhibited by Con-A stimulated SLC from CD rats and NASLC from CD and CA rats. In conclusion, reduced T-lymphocyte proliferation in copper-deficiency: is influenced by the tissue source of lymphoid cells; is not due to impaired ability of T-lymphocytes to proliferate; and may be due to imparied enhancer function for T-lymphocyte proliferation by macrophages." @default.
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- W2010502097 date "1987-02-01" @default.
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- W2010502097 title "Lymphoid cell functions during copper deficiency" @default.
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- W2010502097 doi "https://doi.org/10.1016/s0271-5317(87)80032-5" @default.
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