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• W2010632341 abstract "The potential estrogenicity or antiestrogenicity of Δ9-tetrahydrocannabinol (THC), the chief psychoactive constituent of marijuana, was evaluated in immature female rats treated for 3 days with estradiol (E2; 1 μg/kg), THC (10 mg/kg body weight), or E2 + THC. Estradiol treatment significantly increased anterior pituitary, uterine, and oviduct weights. When THC was administered with E2, it prevented the E2-induced increase in pituitary weight, but had no effect on either the uterine or oviduct weight response to E2. In the E2 treatment group, basal prolactin levels were increased and a prolactin surge occurred on the afternoon of the 26th day of age. However, E2-stimulated basal and surge levels of prolactin were significantly attenuated by concomitant THC treatment. Moreover, pituitary prolactin concentrations, which were elevated in Entreated rats, did not differ from control values in E2 + THC-treated animals. The E2-induced decrease in dopamine turnover rates in the medial basal hypothalamus and increase in the number of anterior pituitary dopamine D2-binding sites (Bmax) were not affected by concomitant THC treatment. Thus, THC antagonizes E2 action on the anterior pituitary via yet to be elucidated mechanism(s)." @default.
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• W2010632341 date "1991-02-01" @default.
• W2010632341 modified "2023-10-17" @default.
• W2010632341 title "Δ9-Tetrahydrocannabinol antagonism of the anterior pituitary response to estradiol in immature female rats" @default.
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• W2010632341 doi "https://doi.org/10.1016/0039-128x(91)90131-e" @default.
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