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- W201093759 endingPage "158" @default.
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- W201093759 abstract "The primary regulator of testicular function is gonadotropin-releasing hormone (GnRH) that is secreted episodically by hypothalamic neurons into the portal vascular system. GnRH stimulates the synthesis and secretion of luteinizing hormone (LH) and follicle-stimulating hormone (FSH), which are produced by gonadotrophs of the anterior pituitary. LH stimulates testosterone synthesis after binding to specific receptors on Leydig cells, whereas Sertoli cells in the seminiferous tubules are the primary target of FSH (1, 2). Accordingly, mass lesions and other disorders of the suprasellar region or pituitary that disrupt GnRH or LH/FSH production result in impaired testicular function that is designated hypogonadotropic hypogonadism because of the deficiency of LH and FSH. These disorders are the subject of this chapter." @default.
- W201093759 created "2016-06-24" @default.
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- W201093759 creator A5065676525 @default.
- W201093759 creator A5077499862 @default.
- W201093759 creator A5079443706 @default.
- W201093759 date "2004-01-01" @default.
- W201093759 modified "2023-09-26" @default.
- W201093759 title "Male Hypogonadism Resulting From Disorders of the Pituitary and Suprasellar Region" @default.
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- W201093759 doi "https://doi.org/10.1007/978-1-59259-727-7_8" @default.
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