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- W2011014909 abstract "The nephrotoxic effects of sodium iopanoate, infused intravenously in dosages of 1.5, 3 and 6 g over a 2-hour period in eight dogs (22–27 kg) in both the hydrated and dehydrated stages, were investigated. On biochemical, hemodynamic and morphological studies, the nephrotoxic effect was found to be clearly dose-dependent and greater in the dehydrated stage. Previously suggested mechanisms for acute renal failure, such as a direct tubular toxicity, tubular obstruction and hypotension, could not be supported as main factors in the development of acute renal failure post-cholecystography, although they might well have a contributory effect. The primary cause of renal failure post-cholecystography cannot be determined unequivocally from this investigation. However, the observed findings would appear to be consistent with a toxic effect of iopanoate in the small vessels possibly resulting in ischemic tubular necrosis." @default.
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- W2011014909 date "1978-05-01" @default.
- W2011014909 modified "2023-09-26" @default.
- W2011014909 title "Nephrotoxicity of Sodium Iopanoate in Hydrated and Dehydrated Dogs" @default.
- W2011014909 doi "https://doi.org/10.1097/00004424-197805000-00012" @default.
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