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• W2011083820 abstract "We investigated the effect of parkin on mitochondrial function and apoptosis in SH-SY5Y, L6, RD, and COS-1 cells. Wild-type parkin attenuated reactive oxygen species (ROS) production in SH-SY5Y cells and mutant parkin enhanced ROS production in SH-SY5Y and L6 cells. Reactive oxygen intermediates, that were detected in mitochondria, were decreased in cells with overexpression of parkin. Parkin prevented apoptosis and enhanced mitochondrial membrane potentials in SH-SY5Y and L6 cells not in COS-1 cells. Expressions and enzymatic activities of mitochondrial respiratory chain complexes were not uniformly enhanced but those of complex 1 were selectively enhanced. The present results suggest the cell-selective function of parkin, i.e., parkin possesses anti-apoptotic and anti-oxidant function in neuronal or myogenic cells but not in kidney cells." @default.
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• W2011083820 date "2006-09-01" @default.
• W2011083820 modified "2023-09-27" @default.
• W2011083820 title "Parkin affects mitochondrial function and apoptosis in neuronal and myogenic cells" @default.
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• W2011083820 doi "https://doi.org/10.1016/j.bbrc.2006.06.201" @default.
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