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• W2011116990 endingPage "624" @default.
• W2011116990 startingPage "615" @default.
• W2011116990 abstract "A fine regulation of the amiloride-sensitive Epithelial Sodium Channel (ENaC), made of alpha, beta and gamma subunits, is crucial for maintenance of Na+ balance and blood pressure. Both beta- and gamma-ENaC participate in negative regulation by interacting with Nedd4-2, an E3 ubiquitin-ligase. Disruption of this interaction results in increased ENaC activity (Liddle syndrome). By two-hybrid screenings, we identified new potential partners of alpha-ENaC: WWP1 (E3 ubiquitin-ligase protein), UBC9 and TSG101 (E2 ubiquitin/SUMO-conjugating enzymes) and confirmed these interactions in GST pull-down assays. All these partners are implicated in protein trafficking and could be involved in the regulation of ENaC activity." @default.
• W2011116990 created "2016-06-24" @default.
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• W2011116990 date "2003-07-01" @default.
• W2011116990 modified "2023-09-25" @default.
• W2011116990 title "Identification of new partners of the epithelial sodium channel alpha subunit" @default.
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• W2011116990 doi "https://doi.org/10.1016/s1631-0691(03)00154-9" @default.
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• W2011116990 hasPublicationYear "2003" @default.
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