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- W2011241929 abstract "Abstract The two major secretion products of the normal testes were measured in peripheral plasma of eight patients with Klinefelter's syndrome; two agonadal, one castrate, one patient with isolated gonadotropin deficiency, and one patient with unclassified primary hypogonadism. Plasma testosterone and 17-α-hydroxyprogesterone were very low in all but the Klinefelter's patients. Testosterone values markedly varied, but were generally subnormal in Klinefelter's, ranging from 25–425 mμg 100 ml , 187 ± 160. The 17-α-hydroxyprogesterone (17-OHP), however, was elevated in three and within the normal range in five [Klinefelter's 120 ± 63 (SD) vs. normal 97 ± 21 (SD) m μg 100 ml , respectively]. The 17-OHP values are inappropriate when considered with the subnormal testosterone values. One of the youngest patients studied had a subnormal testosterone but a plasma 17-OHP that was three times the mean for a normal adult male. The 17-OHP in Klinefelter's was of testicular origin, since dexamethasone had minimal effect, but testosterone almost completely reduced plasma 17-OHP. The marked circadian variation of 17-OHP in normal males was not present in Klinefelter's, although noncyclic fluctuations were observed. LH in the form of chorionic gonadotropin produced a variable response. Some of the patients exhibited a rise in plasma 17-OHP and testosterone despite already elevated 17-OHP levels. The studies indicate the presence of a disorder involving Leydig cell steroidogenesis in Klinefelter's syndrome." @default.
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- W2011241929 date "1973-07-01" @default.
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- W2011241929 title "Leydig cell function in Klinefelter's syndrome" @default.
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- W2011241929 doi "https://doi.org/10.1016/0026-0495(73)90060-7" @default.
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