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• W2011293364 abstract "Toll-like receptors (TLRs) are a family of evolutionarily conserved molecules that directly detect pathogen invasion or tissue damage and initiate a biological response. TLRs can signal through two primary intracellular pathways and as such can induce either immuno-stimulatory or immuno-modulatory molecules. Both sides of this twin-edged sword are being examined for their therapeutic potential in combating neurological disease. The immuno-stimulatory properties of TLRs are being used to generate tumor-specific immune responses to CNS tumors while the immuno-modulatory properties are being used to suppress damaging inflammatory responses to stroke. Recently, a third component of TLR signaling has begun to emerge--that of direct neuroprotection. Hence, the TLRs offer novel targets for the treatment of neurological disease." @default.
• W2011293364 created "2016-06-24" @default.
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• W2011293364 date "2008-02-01" @default.
• W2011293364 modified "2023-09-27" @default.
• W2011293364 title "Toll-like receptors: novel pharmacological targets for the treatment of neurological diseases" @default.
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• W2011293364 doi "https://doi.org/10.1016/j.coph.2007.09.009" @default.
• W2011293364 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2674015" @default.
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