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- W2011511235 abstract "Preceding investigations have demonstrated, in general, a decrease in cardiac output and an increase in peripheral resistance after the administration of ganglionic blocking agents.1–3 Mecamylamine has been shown to have hemodynamic effects similar to ganglionic blocking agents used previously.4 It has not been established, however, whether the effects of these agents are due purely to the reduction in cardiac filling pressure secondary to peripheral pooling of blood,3–5 or whether additional factors, such as a decreased discharge from the sympathetic nervous system, are significant as a mechanism of the decreased output.6 Submersion of the human subject in water after the administration of hexamethonium bromide restored the arterial blood pressure nearly to control values, but no data on cardiac output was recorded during this experiment.7 In the dog heart-lung preparation or isolated papillary muscle strip, hexamethonium produces an increased force of myocardial contraction; hence, it seems that a direct depressant effect on the myocardium is not likely from this agent.8,9 Mecamylamine, on the other hand, is reported to decrease contractility of heart muscle strips in vitro.10 The present report describes a series of experiments designed to test the hemodynamic effect of increasing the central venous pressure after the production of ganglionic blockade." @default.
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- W2011511235 date "1960-11-01" @default.
- W2011511235 modified "2023-09-23" @default.
- W2011511235 title "Investigation of the mechanism of the hemodynamic effects of ganglionic blockade produced by mecamylamine" @default.
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- W2011511235 doi "https://doi.org/10.1016/0002-8703(60)90360-4" @default.
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