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• W2012022071 abstract "The present study investigates the possible mechanism responsible for the neurotoxicity of d,l-homocysteine in primary culture of rat cerebellar granule cells. Neurotoxicity was assessed by measuring the amount of lactate dehydrogenase released from the cells following homocysteine treatment. d,l-Homocysteine (> 300 μM; 16–22 h) induced the release of lactate dehydrogenase from the cells in a concentration-dependent manner. The N-methyl-d-aspartate (NMDA) antagonist (±)-2-amino-5-phosphonopentanoic acid (APV) partially blocked the homocysteine-mediated neurotoxicity. However, the non-NMDA receptor antagonist 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX) did not block the homocysteine-mediated toxicity. The homocysteine-mediated neurotoxicity was mostly prevented by the co-administration of superoxide dismutase and catalase or catalase alone. The results suggest that homocysteine induces neuronal cell death by stimulating NMDA receptor as well as by producing free radicals." @default.
• W2012022071 created "2016-06-24" @default.
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• W2012022071 date "1996-09-01" @default.
• W2012022071 modified "2023-10-15" @default.
• W2012022071 title "Involvement of N-methyl-d-aspartate receptor and free radical in homocysteine-mediated toxicity on rat cerebellar granule cells in culture" @default.
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• W2012022071 doi "https://doi.org/10.1016/0304-3940(96)13011-1" @default.
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