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• W2012023622 abstract "Allergen‐induced airway inflammation may lead to allergic asthma, a chronic inflammatory disease of the respiratory system. Despite its high incidence, the majority of the world's population is unaffected by allergic airway inflammation most likely due to innate protective mechanism(s) in the respiratory system. The mammalian airway epithelia constitutively express uteroglobin (UG), a protein with potent anti‐inflammatory and anti‐chemotactic properties. We report here that UG binds to FPR2, a G‐protein coupled receptor, inhibits chemotaxis, down‐regulates SOCS‐3 gene expression and STAT‐1 activation, which are critical for the differentiation of T‐helper 2 (T H 2) cells that secrete pro‐inflammatory T H 2 cytokines. We propose that UG suppresses allergen‐mediated activation of T H 2 response by down‐regulating the expression of genes that are critical for T H 2 differentiation." @default.
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• W2012023622 date "2006-10-05" @default.
• W2012023622 modified "2023-10-13" @default.
• W2012023622 title "Uteroglobin suppresses allergen-induced T_H2 differentiation by down-regulating the expression of serum amyloid A and SOCS-3 genes" @default.
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• W2012023622 doi "https://doi.org/10.1016/j.febslet.2006.09.059" @default.
• W2012023622 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1859844" @default.
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