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• W2012037560 endingPage "377" @default.
• W2012037560 startingPage "367" @default.
• W2012037560 abstract "Adipose tissue controls whole-body lipid flux, thereby modulating both glucose and lipid homeostasis in humans. Discovery of new targets that regulate fatty acids in adipocytes might lead to therapeutic modalities that can prevent insulin resistance and type 2 diabetes. Acquired resistance to the action of insulin to stimulate glucose transport in skeletal muscle is associated with obesity and promotes the development of type 2 diabetes. In skeletal muscle, insulin resistance can result from high levels of circulating fatty acids that disrupt insulin signalling pathways. However, the severity of insulin resistance varies greatly among obese people. Here we postulate that this variability might reflect differences in levels of lipid-droplet proteins that promote the sequestration of fatty acids within adipocytes in the form of triglycerides, thereby lowering exposure of skeletal muscle to the inhibitory effects of fatty acids." @default.
• W2012037560 created "2016-06-24" @default.
• W2012037560 creator A5015721009 @default.
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• W2012037560 creator A5082591544 @default.
• W2012037560 creator A5085687728 @default.
• W2012037560 date "2008-05-01" @default.
• W2012037560 modified "2023-10-18" @default.
• W2012037560 title "Adipocyte dysfunctions linking obesity to insulin resistance and type 2 diabetes" @default.
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