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• W2012132325 abstract "Plant mitochondria generate nitric oxide (NO) under anoxia through the action of cytochrome c oxidase and other electron transport chain components on nitrite. This reductive mechanism operates under aerobic conditions at high electron transport rates. Indirect evidence also indicates that the oxidative pathway of NO production may be associated with mitochondria. We review the consequences of mitochondrial NO production, including the inhibition of oxygen uptake by cytochrome c oxidase, the inhibition of aconitase and succinate dehydrogenase, the induction of alternative oxidase, and the nitrosylation of several proteins, including glycine decarboxylase. The importance of these events in adaptation to abiotic and biotic stresses is discussed." @default.
• W2012132325 created "2016-06-24" @default.
• W2012132325 creator A5012989382 @default.
• W2012132325 creator A5019754635 @default.
• W2012132325 creator A5082173447 @default.
• W2012132325 date "2014-11-01" @default.
• W2012132325 modified "2023-10-03" @default.
• W2012132325 title "Plant mitochondria: Source and target for nitric oxide" @default.
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• W2012132325 doi "https://doi.org/10.1016/j.mito.2014.02.003" @default.
• W2012132325 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24561220" @default.
• W2012132325 hasPublicationYear "2014" @default.
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