FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2012204755> ?p ?o ?g. }

• W2012204755 endingPage "54" @default.
• W2012204755 startingPage "48" @default.
• W2012204755 abstract "Abstract: We characterized the expression levels of the retinoid Z receptor α (RZR α ), ROR α mRNA isoforms (ROR α 1, ROR α 2, and ROR α 3), and both melatonin receptor MT 1 and hydroxindole‐ O ‐methyltransferase (HIOMT) genes. For this purpose, the following human peripheral blood mononuclear cells populations were isolated: monocytes (CD14 + cells), B lymphocytes (CD19 + cells), T helper lymphocytes (CD14 − CD4 + ), cytotoxic T lymphocytes (CD56 − CD8 + cells), and natural killer (NK) lymphocytes (CD56 + cells). PBMCs subsets were obtained by Dynabeads M‐450 (Dynal) isolation procedure. We observed a strong gene expression signal for RZR α in all subpopulations studied, whereas both ROR α 1 and ROR α 2 transcripts were amplified only in CD8 + cells. Specific signal for ROR α 2 was obtained in all subpopulations studied, but we were not able to detect the ROR α 3 mRNA transcript in human immune cells studied. A weaker signal (especially in CD19 + cells) was also detected in all subsets of cells for the MT 1 gene. With regard to HIOMT, a strong signal was achieved among all but one subpopulation of cells; the only exception was CD14 + cells. Thus, in addition to its classical function in the nervous and endocrine system, melatonin could act directly as a paracrine and/or autocrine agent in the human immune system." @default.
• W2012204755 created "2016-06-24" @default.
• W2012204755 creator A5017023675 @default.
• W2012204755 creator A5017730925 @default.
• W2012204755 creator A5022305364 @default.
• W2012204755 creator A5067897234 @default.
• W2012204755 date "2004-04-08" @default.
• W2012204755 modified "2023-10-18" @default.
• W2012204755 title "mRNA expression of nuclear receptor RZR/RORalpha, melatonin membrane receptor MT1, and hydroxindole-O-methyltransferase in different populations of human immune cells" @default.
• W2012204755 cites W1502098090 @default.
• W2012204755 cites W1526473725 @default.
• W2012204755 cites W1528384739 @default.
• W2012204755 cites W1540874939 @default.
• W2012204755 cites W1562048214 @default.
• W2012204755 cites W1570607043 @default.
• W2012204755 cites W1589377963 @default.
• W2012204755 cites W1614960091 @default.
• W2012204755 cites W1910220224 @default.
• W2012204755 cites W1966356185 @default.
• W2012204755 cites W1977332155 @default.
• W2012204755 cites W1986615746 @default.
• W2012204755 cites W1988148616 @default.
• W2012204755 cites W1989418616 @default.
• W2012204755 cites W1994290678 @default.
• W2012204755 cites W1996148507 @default.
• W2012204755 cites W1999529148 @default.
• W2012204755 cites W2002226670 @default.
• W2012204755 cites W2003532302 @default.
• W2012204755 cites W2007964356 @default.
• W2012204755 cites W2009745241 @default.
• W2012204755 cites W2015542694 @default.
• W2012204755 cites W2016868037 @default.
• W2012204755 cites W2021399303 @default.
• W2012204755 cites W2025584412 @default.
• W2012204755 cites W2030848908 @default.
• W2012204755 cites W2032779870 @default.
• W2012204755 cites W2033336970 @default.
• W2012204755 cites W2034348289 @default.
• W2012204755 cites W2047224082 @default.
• W2012204755 cites W2048884799 @default.
• W2012204755 cites W2050309531 @default.
• W2012204755 cites W2051121914 @default.
• W2012204755 cites W2051603449 @default.
• W2012204755 cites W2054173875 @default.
• W2012204755 cites W2059352599 @default.
• W2012204755 cites W2059576405 @default.
• W2012204755 cites W2061415106 @default.
• W2012204755 cites W2062196582 @default.
• W2012204755 cites W2062708982 @default.
• W2012204755 cites W2064064502 @default.
• W2012204755 cites W2064280448 @default.
• W2012204755 cites W2064281169 @default.
• W2012204755 cites W2068820723 @default.
• W2012204755 cites W2074658037 @default.
• W2012204755 cites W2079071895 @default.
• W2012204755 cites W2092481580 @default.
• W2012204755 cites W2093769918 @default.
• W2012204755 cites W2111561559 @default.
• W2012204755 cites W2112916976 @default.
• W2012204755 cites W2127228129 @default.
• W2012204755 cites W2128838531 @default.
• W2012204755 cites W2131962483 @default.
• W2012204755 cites W2135167537 @default.
• W2012204755 cites W2136350449 @default.
• W2012204755 cites W2141293774 @default.
• W2012204755 cites W2155679235 @default.
• W2012204755 cites W2157753912 @default.
• W2012204755 cites W2160692145 @default.
• W2012204755 cites W2162568193 @default.
• W2012204755 cites W2163789824 @default.
• W2012204755 cites W2184742230 @default.
• W2012204755 cites W4229807268 @default.
• W2012204755 cites W4234033758 @default.
• W2012204755 cites W4240999623 @default.
• W2012204755 doi "https://doi.org/10.1111/j.1600-079x.2004.00135.x" @default.
• W2012204755 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/15230868" @default.
• W2012204755 hasPublicationYear "2004" @default.
• W2012204755 type Work @default.
• W2012204755 sameAs 2012204755 @default.
• W2012204755 citedByCount "103" @default.
• W2012204755 countsByYear W20122047552012 @default.
• W2012204755 countsByYear W20122047552013 @default.
• W2012204755 countsByYear W20122047552014 @default.
• W2012204755 countsByYear W20122047552015 @default.
• W2012204755 countsByYear W20122047552016 @default.
• W2012204755 countsByYear W20122047552017 @default.
• W2012204755 countsByYear W20122047552018 @default.
• W2012204755 countsByYear W20122047552019 @default.
• W2012204755 countsByYear W20122047552020 @default.
• W2012204755 countsByYear W20122047552021 @default.
• W2012204755 countsByYear W20122047552022 @default.
• W2012204755 countsByYear W20122047552023 @default.
• W2012204755 crossrefType "journal-article" @default.
• W2012204755 hasAuthorship W2012204755A5017023675 @default.
• W2012204755 hasAuthorship W2012204755A5017730925 @default.
• W2012204755 hasAuthorship W2012204755A5022305364 @default.
• W2012204755 hasAuthorship W2012204755A5067897234 @default.
• W2012204755 hasConcept C104317684 @default.

• next