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- W2012302647 abstract "Abstract At the core of eukaryotic aerobic life, mitochondrial function like ‘hubs’ in the web of energetic and redox processes in cells. In the heart, these networks—extending beyond the complex connectivity of biochemical circuit diagrams and apparent morphology—exhibit collective dynamics spanning several spatiotemporal levels of organization, from the cell, to the tissue, and the organ. The network function of mitochondria, i.e., mitochondrial network energetics , represents an advantageous behavior. Its coordinated action, under normal physiology, provides robustness despite failure in a few nodes, and improves energy supply toward a swiftly changing demand. Extensive diffuse loops, encompassing mitochondrial–cytoplasmic reaction/transport networks, control and regulate energy supply and demand in the heart. Under severe energy crises, the network behavior of mitochondria and associated glycolytic and other metabolic networks collapse, thereby triggering fatal arrhythmias. WIREs Syst Biol Med 2012, 4:599–613. doi: 10.1002/wsbm.1188 This article is categorized under: Analytical and Computational Methods > Analytical Methods Models of Systems Properties and Processes > Cellular Models Physiology > Mammalian Physiology in Health and Disease" @default.
- W2012302647 created "2016-06-24" @default.
- W2012302647 creator A5020291322 @default.
- W2012302647 creator A5036417376 @default.
- W2012302647 date "2012-08-15" @default.
- W2012302647 modified "2023-09-23" @default.
- W2012302647 title "Mitochondrial network energetics in the heart" @default.
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