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- W2012504815 abstract "Among complex disorders, those concerning neuropsychiatric phenotypes involve particular challenges compared to disorders with more easily distinguished clinical signs and measures. One such common and unusually challenging phenotype to disentangle genetically is developmental dyslexia (DD), or reading disability, defined as the inability to learn to read and write for an otherwise normally intelligent child with normal senses and educational opportunity. There is presently ample evidence for the strongly biological etiology for DD, and a dozen susceptibility genes have been suggested. Many of these genes point to common but previously unsuspected biological mechanisms, such as neuronal migration and cilia functions. I discuss here the state-of-the-art in genomic and neurobiological aspects of DD research, starting with short general background to its history." @default.
- W2012504815 created "2016-06-24" @default.
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- W2012504815 date "2014-09-01" @default.
- W2012504815 modified "2023-10-07" @default.
- W2012504815 title "The molecular genetics and neurobiology of developmental dyslexia as model of a complex phenotype" @default.
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- W2012504815 doi "https://doi.org/10.1016/j.bbrc.2014.07.102" @default.
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