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- W2012591801 abstract "Both systemic and central injections of glucose can enhance memory. For example, glucose reverses impairments on inhibitory avoidance resulting from intra-amygdala injections of morphine. The present experiment investigated the ability of glucose to reverse memory impairments resulting from intra-amygdala injections of propranolol, a beta-noradrenergic antagonist. Pretraining administration of 10 microg propranolol significantly reduced inhibitory avoidance retention latencies but had no effect on performance in a spontaneous alternation task. Coadministration of glucose into the amygdala at 3 doses (1.5, 3.0, and 6.0 microg) did not reverse the propranolol-induced inhibitory avoidance deficits. However, coadministration of 2.5 microg of glutamate with the propranolol did reverse these deficits. The ability of glucose to reverse impairments following intra-amygdala injections of morphine but not propranolol may reflect the neurotransmitter system or systems through which glucose exerts its effects." @default.
- W2012591801 created "2016-06-24" @default.
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- W2012591801 date "1996-01-01" @default.
- W2012591801 modified "2023-09-26" @default.
- W2012591801 title "Inhibitory avoidance impairments induced by intra-amygdala propranolol are reversed by glutamate but not glucose.'" @default.
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- W2012591801 doi "https://doi.org/10.1037/0735-7044.110.5.1033" @default.
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