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- W2012597327 abstract "IN PREVIOUS studies it has been demonstrated that gastnc ulceratron, produced in mice by rmmobrhzatron stress, is paralleled m effect and severity by eqmvalent penods of food deprrvatron wrthout rmmobrhzatron [I]. These data have also indicated that gastric pathology resulting from food depnvatron alone, m mace, was an inverse function of gastric food retention for the duration of the depnvatron perrod. The latter variable and, by corollary, the former, appeared to be consrstently dependent upon age and werght, and perhaps to some extent to drfferences in the stress conditions themselves durmg the 24-hr perrod m which the gastrrc lessons develop. Food deprrvatron has been assumed to be a “stress” for rodents [2]; however, thus, as a behavioral construct alone, falls to satrsfactorally explain all of our previous findmgs [3]. Ammals subjected to rmmobrhzatron stress are, by methodologrcal definition, Isolated durmg the “stress” Interval; whereas food depnvatron may be mmated whether ammals are isolated or grouped during the “food-deprrvatron mterval”. Since rsolatron has been referred to as a possible “emotronal stressor” [4] and may, as such, therefore independently contribute to the etrology of gastnc lessons, It was the purpose of this study to Investigate the drfferentral effects of rsolatron and aggregation upon the development of gastric pathology m mice. Smce the duration of aggregation or isolatron may serve as an unaccounted factor, this variable was consrdered by utrhzmg two discrepant penods (8 and 30 days) with age and duration of food deprivation maintained constant." @default.
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- W2012597327 date "1966-09-01" @default.
- W2012597327 modified "2023-10-16" @default.
- W2012597327 title "Isolation-induced facilitation of gastric ulcerogenesis in mice" @default.
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- W2012597327 doi "https://doi.org/10.1016/0022-3999(66)90061-4" @default.
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