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• W2012643403 abstract "Cellular immune responses are known to be influenced by gene products of the major histocompatibility complex (MHC). Cell surface antigens encoded by the I-A subregion of the MHC are critical in antigen processing and restricting CD4+ helper T-cell interactions. Antibodies directed against such antigens have been shown to modulate immune responses in vitro and in vivo. Anti-I-A antibody therapy has been shown to block antigen-specific T-cell activation, and to promote the development of antigen-specific T-suppressor cells. When applied to models of autoimmunity and allograft rejection in laboratory animals, anti-I-A antibody therapy has resulted in consistent and significant alterations in the natural history of these processes. Clinical application of this therapy to human disease has been inconsistent and disappointing. Before proceeding with further clinical trials, it will be important to standardize such antibodies with regard to isotype as well as antigen affinity and specificity." @default.
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• W2012643403 date "1988-12-01" @default.
• W2012643403 modified "2023-09-26" @default.
• W2012643403 title "The role of anti-I-A antibody therapy in the management of autoimmune disease and in transplantation" @default.
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• W2012643403 doi "https://doi.org/10.1016/0169-409x(88)90015-4" @default.
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