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- W2012676747 abstract "• Three South African blacks with hepatocellular carcinoma and arterial hypertension are described. Plasma angiotensinogen (renin substrate) concentrations were increased eightfold to 10-fold in the two patients in whom these concentrations were measured. One of these two patients also showed a 34-fold rise in plasma inactive, active, and total renin concentrations, and an elevated plasma renin activity (2.73 ng·L<sup>−1</sup>·s<sup>−1</sup>angiotensin l/mL/h). Inactive renin (prorenin) constituted 90% of the total plasma renin concentration. In the third patient only plasma renin activity was measured, and this was considerably raised (6.05 ng·L<sup>−1</sup>·s<sup>−1</sup>; angiotensin l/mL/h). Thus, the arterial hypertension that rarely complicates hepatocellular carcinoma may be caused either by a combination of eutopic synthesis of excessive quantities of angiotensinogen and ectopic production and secretion of active renin by malignant hepatocytes, or by eutopic production of angiotensinogen alone. (<i>Arch Intern Med</i>. 1989;149:2111-2113)" @default.
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- W2012676747 date "1989-09-01" @default.
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- W2012676747 title "Arterial Hypertension as a Paraneoplastic Phenomenon in Hepatocellular Carcinoma" @default.
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- W2012676747 doi "https://doi.org/10.1001/archinte.1989.00390090135028" @default.
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