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- W2013115334 abstract "Long-term potentiation (LTP) of synaptic strength occurs during learning and can last for long periods, making it a probable mechanism for memory storage. LTP induction results in calcium entry, which activates calcium/calmodulin-dependent protein kinase II (CaMKII). CaMKII subsequently translocates to the synapse, where it binds to NMDA-type glutamate receptors and produces potentiation by phosphorylating principal and auxiliary subunits of AMPA-type glutamate receptors. These processes are all localized to stimulated spines and account for the synapse-specificity of LTP. In the later stages of LTP, CaMKII has a structural role in enlarging and strengthening the synapse." @default.
- W2013115334 created "2016-06-24" @default.
- W2013115334 creator A5031978385 @default.
- W2013115334 creator A5071574342 @default.
- W2013115334 creator A5075016550 @default.
- W2013115334 date "2012-02-15" @default.
- W2013115334 modified "2023-10-10" @default.
- W2013115334 title "Mechanisms of CaMKII action in long-term potentiation" @default.
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