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- W2013459541 abstract "Diabetic nephropathy (DN) is the major cause of end-stage renal disease in Western countries and its prevalence continues to increase (United States Renal Data System 2010, http://www.usrds.org/). Treatments currently utilised for DN provide only partial renoprotection, hence the need to identify new targets for therapeutic intervention. Metabolic and haemodynamic abnormalities have been implicated in the pathogenesis of DN, triggering the activation of intracellular signaling molecules that lead to the dysregulation of vascular growth factors and cytokines, such as vascular endothelial growth factor (VEGF) and angiopoietins, important players in the functional and structural regulation of the glomerular filtration barrier. This review focuses on the importance of VEGF-A and angiopoietins in kidney physiology and in the diabetic kidney, exploring their potential therapeutic role in the prevention and delay of diabetic glomerulopathy." @default.
- W2013459541 created "2016-06-24" @default.
- W2013459541 creator A5032861135 @default.
- W2013459541 creator A5057777814 @default.
- W2013459541 date "2012-12-01" @default.
- W2013459541 modified "2023-09-27" @default.
- W2013459541 title "VEGF and angiopoietins in diabetic glomerulopathy: How far for a new treatment?" @default.
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- W2013459541 doi "https://doi.org/10.1016/j.metabol.2012.04.004" @default.
- W2013459541 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/22554833" @default.
- W2013459541 hasPublicationYear "2012" @default.
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