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- W2013525882 abstract "Salmonella enterica is responsible for a large number of diseases in a wide-range of hosts. Two of the global regulators involved in controlling gene expression during the infection cycle of the bacterium are Fur and HNS. In this paper, we demonstrate computationally that Fur and HNS have disproportionately high density of binding sites in the Pathogenicity Islands on the Salmonella chromosome. Moreover, the frequency of binding sites for the two proteins is correlated throughout the genome of the organism. These results indicate a complex interplay between Fur and HNS in regulating cellular global behavior." @default.
- W2013525882 created "2016-06-24" @default.
- W2013525882 creator A5079026550 @default.
- W2013525882 creator A5083112582 @default.
- W2013525882 date "2012-11-01" @default.
- W2013525882 modified "2023-10-14" @default.
- W2013525882 title "Interplay between Fur and HNS in controlling virulence gene expression in Salmonella typhimurium" @default.
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- W2013525882 doi "https://doi.org/10.1016/j.compbiomed.2012.09.005" @default.
- W2013525882 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/23040276" @default.