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- W2013556987 abstract "In this review, we discuss how protozoan parasites alter immature and mature B cell compartment. B1 and marginal zone (MZ) B cells, considered innate like B cells, are activated during protozoan parasite infections, and they generate short lived plasma cells providing a prompt antibody source. In addition, protozoan infections induce massive B cell response with polyclonal activation that leads to hypergammaglobulnemia with serum antibodies specific for the parasites and self and/or non related antigens. To protect themselves, the parasites have evolved unique ways to evade B cell immune responses inducing apoptosis of MZ and conventional mature B cells. As a consequence of the parasite induced-apoptosis, the early IgM response and an already establish humoral immunity are affected during the protozoan parasite infection. Moreover, some trypanosomatides trigger bone marrow immature B cell apoptosis, influencing the generation of new mature B cells. Simultaneously with their ability to release antibodies, B cells produce cytokines/quemokines that influence the characteristic of cellular immune response and consequently the progression of parasite infections." @default.
- W2013556987 created "2016-06-24" @default.
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- W2013556987 date "2012-01-01" @default.
- W2013556987 modified "2023-10-12" @default.
- W2013556987 title "B-Cell Response during Protozoan Parasite Infections" @default.
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- W2013556987 doi "https://doi.org/10.1155/2012/362131" @default.
- W2013556987 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3270435" @default.
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