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- W2013851063 abstract "Exogenous administration of testosterone produced several metabolic tissue-specific changes in female mouse kidneys, but not in the liver. The hormone induced ornithine decarboxylase (ODC) activity, and also profoundly influenced metabolism of S-adenosylmethionine (AdoMet). Therefore, the activity of the AdoMet-synthesizing enzyme (AdoMet synthetase) and of cystathionine synthase, which commits homocysteine irreversibly to the transsulfuration pathway, were significantly increased. In contrast to the level of AdoMet in the liver the renal level of this metabolite was augmented, whereas the level of S-adenosylhomocysteine (AdoHcy) did not change. This resulted in an increase of the AdoMet/AdoHcy) ratio. In testosterone-treated mice, pulse-labelled with [metyl-14C]methionine, the radioactivity recovered in the kidneys doubled, but in the liver remained the same. The rise in radioactivity recovered occured mainly in TCA-soluble compounds and lipids, and to a smaller extent, in proteins and nucleic acids." @default.
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- W2013851063 date "1992-04-01" @default.
- W2013851063 modified "2023-10-17" @default.
- W2013851063 title "Tissue-specific effects of testosterone on S-adenosylmethionine formation and utilization in the mouse" @default.
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- W2013851063 doi "https://doi.org/10.1016/0304-4165(92)90113-9" @default.
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